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Journal of Case Reports in Medicine
An Open Access Journal for Case Reports in Medicine
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![Figure 1: Bone marrow morphology and G-banding revealed clonal evolution. (a) Bone marrow smear (Wright- Giemsa, 1,000×) showed MDS at diagnosis. (b) Karyotype at MDS diagnosis was 46,XY,del(5)(q?)[1]/45,XY,idem, -7,-14,-17,+mar1,+mar2[7]/45,XY,-5[4]/46,XY[8]. A major abnormal clone is shown in Figure 1(b). (c) Bone marrow smear (Wright-Giemsa, 1,000×) showed AML with multilineage dysplasia upon progression of anemia 4 months after MDS diagnosis. The size of the blast cells ranged from medium to large; they](https://jocrm.org/sites/default/files/styles/large/public/JCaseRepMed%2C%202013_2_1_105.png?itok=iqd6mGVA "Figure 1: Bone marrow morphology and G-banding revealed clonal evolution. (a) Bone marrow smear (Wright- Giemsa, 1,000×) showed MDS at diagnosis. (b) Karyotype at MDS diagnosis was 46,XY,del(5)(q?)[1]/45,XY,idem, -7,-14,-17,+mar1,+mar2[7]/45,XY,-5[4]/46,XY[8]. A major abnormal clone is shown in Figure 1(b). (c) Bone marrow smear (Wright-Giemsa, 1,000×) showed AML with multilineage dysplasia upon progression of anemia 4 months after MDS diagnosis. The size of the blast cells ranged from medium to large; they showed a high nuclear:cytoplasmic ratio and visible nucleoli. (d) Karyotypes at diagnosis of transformation to AML was 45,XY,del(5)(q?),-7,t(9;22)(q34;q11.2),-14,-17, +mar1,mar2[20]. The Philadelphia chromosome is marked by an arrowhead.")
